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NPTEL Video Lectures, IIT Video Lectures Online, NPTEL Youtube Lectures, Free Video Lectures, NPTEL Online Courses, Youtube IIT Videos NPTEL Courses. Computer Science Engineering - CSE Class Notes, Engineering Class handwritten notes, exam notes, previous year questions, PDF free download. Find one of your fellow class mates to see if there is something in these notes that wasn’t covered in class. Because I want these notes to provide some more examples for you to read through, I don’t always work the same problems in class as those given in the notes. Likewise, even.

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  1. Hypoxia = inadequate oxygenation of tissue (same definition of a shock). Need O2 for oxidation phosphorylation pathway – where you get ATP from inner Mito membrane (electron transport system, called oxidative phosphorylation). The last rxn is O2 to receive the electrons. Protons are being kicked off, go back into the membrane, and form ATP, and ATP informed in the mitochondria
  2. Terms:
  3. Oxygen content = Hb x O2 satn + partial pressure of arterial oxygen (these are the 3 main things that carry O2 in our blood)

In Hb, the O2 attaches to heme group (O2 sat’n) Partial pressure of arterial O2 is O2 dissolved in plasma In RBC, four heme groups (Fe must be +2; if Fe+ is +3, it cannot carry O2) Therefore, when all four heme groups have an O2 on it, the O2 sat’n is 100%.

  1. O2 sat’n is the O2 IN the RBC is attached TO the heme group = (measured by a pulse oximeter)
  2. Partial pressure of O2 is O2 dissolved in PLASMA

O2 flow: from alveoli through the interphase, then dissolves in plasma, and increases the partial pressure of O2, diffuses through the RBC membrane and attaches to the heme groups on the RBC on the Hb, which is the O2 sat’n

Therefore – if the partial pressure of O2 is decreased, O2 sat’n HAS to be decreased (B/c O2 came from the amount that was dissolved in plasma)

  1. Causes of tissue hypoxia:
  2. Ischemia (decrease in ARTERIAL blood flow ……NOT venous)

MCC Ischemia is thrombus in the muscular artery (b/c this is the mcc death in USA = MI, therefore MI is a good example of ischemia b/c thrombus is blocking arterial blood flow, producing tissue hypoxia)

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Other causes of tissue ischemia: decrease in Cardiac Output (leads to hypovolemia and cardiogenic shock) b/c there is a decrease in arterial blood flow.

  1. 2 nd MCC of tissue hypoxia = hypoxemia

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Hypoxia = ‘big’ term

Hypoxemia = cause of hypoxia (they are not the same); deals with the partial pressure of arterial O2 (O2 dissolved in arterial plasma, therefore, when the particle pressure of O2 is decreased, this is called hypoxemia). Here are 4 causes of hypoxemia:

  1. Resp acidosis (in terms of hypoxemia) – in terms of Dalton’s law, the sum of the partial pressure of gas must = 760 at atmospheric pressure (have O2, CO2, and nitrogen; nitrogen remains constant – therefore, when you retain CO2, this is resp acidosis; when CO2 goes up, pO2 HAS to go down b/c must have to equal 760; Therefore, every time you have resp acidosis, from ANY cause, you have hypoxemia b/c low arterial pO2; increase CO2= decrease pO2, and vice versa in resp alkalosis).
  2. Ventilation defects – best example is resp distress syndrome (aka hyaline membrane dz in children). In adults, this is called Adult RDS, and has a ventilation defect. Lost ventilation to the alveoli, but still have perfusion; therefore have created an intrapulmonary shunt. Exam question: pt with hypoxemia, given 100% of O2 for 20 minutes, and pO2 did not increase, therefore indicates a SHUNT, massive ventilation defect.
  3. Perfusion defects – knock off blood flow

MCC perfusion defect = pulmonary embolus, especially in prolonged flights, with sitting down and not getting up.

Stasis in veins of the deep veins leads to propagation of a clot and 3-5 days later an embolus develops and embolizes.

In this case, you have ventilation, but no perfusion; therefore there is an increase in dead space. If you give 100% O2 for a perfusion defect, pO2 will go UP (way to distinguish vent from perfusion defect), b/c not every single vessel in the lung is not perfused.

Therefore, perfusion defects because of an increase in dead space, while ventilation defects cause intrapulmonary shunts.

To tell the difference, give 100% O2 and see whether the pO2 stays the same, i.e does not go up (shunt) or increases (increase in dead space).

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Some of the lecture notes were prepared in LaTeX by Alan Dunn, a former MIT student. He used Prof. Zahn's handwritten notes to produce them.

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H/M = Haus, Hermann A., and James R. Melcher. Electromagnetic Fields and Energy. Englewood Cliffs, NJ: Prentice-Hall, 1989. ISBN: 9780132490207.

Lecture notes files.
SES #TOPICSLECTURE NOTES
I. Maxwell's equations
L1

Coulomb-Lorentz force law; Maxwell's equations in integral form; simple electric and magnetic field solutions using Gauss' and Ampere's laws for point, line, and surface charges and currents; superposition; simple cylindrical and spherical source problems

Demos: H/M 10.2.1 - Edgerton's Boomer

(PDF)
L2Derive boundary conditions; apply boundary conditions to surface charge and surface current problems(PDF)
L3

Divergence and Stokes' theorems; Maxwell's equations in differential form; electroquasistatics and magnetoquasistatics (MQS); potential and the gradient operator

Demo: H/M 10.0.1 nonuniqueness of voltage in an MQS system

(PDF)
L4The electric field, electric scalar potential, and the gradient; Poisson's and Laplace's equations; potential of point charge; Coulomb superposition integral(PDF)
L5Method of images(PDF)
L6-L7

Lecture 6

Media: dielectric, conducting, and magnetic constitutive laws; charge relaxation

Demos: H/M 6.6.1 artificial dielectric; 9.4.1 measurement of B-H characteristic (magnetic hysteresis loop)

Lecture 7

Conservation of charge boundary condition; Maxwell capacitor; magnetic dipoles and circuits; reluctance

(PDF - 1.2 MB)
II. Plane waves
L8Wave equation; Poynting's theorem(PDF)
L9Oblique incidence on a perfect conductor; transverse magnetic (TM) waves with oblique incidence on lossless media described by ε and µ; reflection and transmission; transverse electric (TE) waves with oblique incidence on lossless media(PDF)
III. Transmission lines and waveguides
L10

Parallel plate transmission lines; wave equation; sinusoidal steady state

Demo: H/M 13.1.1 visualization of standing waves

(PDF)
L11

Gamma plane; Smith chart; voltage standing wave ratio (VSWR); λ/4 transformer

Demo: V(z,t), I(z,t) movies

(PDF - 2.6 MB)
L12-L13

Lecture 12

Wave equations (lossless); transient waves on transmission lines

Demo: H/M 14.4.1 transmission line matching, reflection, and quasistatic charging

Lecture 13

Reflections from ends; driven and initial value problems

(PDF - 2.5 MB)
L14Rectangular waveguides; TM and TE modes; cut-off(PDF)
IV. Fields and forces
L15

Dielectric waveguides

Demo: evanescent waves

(PDF)
L16-L17

Lecture 16

Energy in electric and magnetic fields; principle of virtual work to find electric and magnetic forces; magnetic circuit problems

Demo: H/M 11.6.2 force on a dielectric material (video)

Lecture 17

Synchronous rotating machines

Film: Synchronous Machines

(PDF)
L18

Self-excited electric and magnetic machines

Demo: H/M 7.7.1 van de Graaff and Kelvin generators (video); self-excited commutator machines

(PDF - 1.3 MB)
V. Antennas and radiation
L19Radiation by charges and currents; setting the gauge; Lorentz gauge; superposition integral solutions for scalar and vector potentials; radiation from a point electric dipole; receiving antenna properties(PDF)
L20

2 element array; broad side and end-fire arrays

Demo: radiation patterns

(PDF - 1.1 MB)
L21Transmitting and receiving antennas; wireless and optical communications(PDF - 1.5 MB)
VI. Acoustics
L22Acoustic waves(PDF - 2.5 MB)
L23Course review(PDF - 1.2 MB)

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